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doi: 10.1007/S10926-022-10075-2 236 BarbaraCClavarioPDe MarzoVLottiRGuglielmiGPorcileAet al

A key finding of our study is the significant reduction of citric acid levels in EP, likely reflecting impaired mitochondrial function and dysregulated energy metabolism

Over longer time periods, namely after 4-OHT induction at E13.5 or E15.5, GFP-negative ECs replace the Bmx-CreERT2 -labeled GFP + cells in submucosal and smaller (distal) mesenteric arteries so that recombined ECs reside only in the larger caliber arteries (Supplementary Fig

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